David O. Walterhouse, MD

Clinical and Community Trials

Biography

Section Head, Oncology, Ann & Robert H. Lurie Children’s Hospital of Chicago
Richard A. Perritt, MD Professorship in Cancer and Blood Disorders, Ann & Robert H. Lurie Children’s Hospital of Chicago
Professor of Pediatrics (Hematology, Oncology, and Stem Cell Transplantation), Northwestern University Feinberg School of Medicine

See Lurie Children's Provider Profile

Education and Background

Fellowship in Pediatric Hematology Oncology, Children's Hospital of Philadelphia 1986-1989
Residency in Pediatrics, Children's Hospital of Philadelphia 1983-1986
MD, University of Michigan Medical School 1983

Clinical Studies

Recruiting

Trastuzumab Deruxtecan for the Treatment of HER2+ Newly Diagnosed or Recurrent Osteosarcoma

Recruiting

Thoracotomy Versus Thoracoscopic Management of Pulmonary Metastases in Patients With Osteosarcoma

Recruiting

Targeted Therapy Directed by Genetic Testing in Treating Pediatric Patients With Relapsed or Refractory Advanced Solid Tumors, Non-Hodgkin Lymphomas, or Histiocytic Disorders (The Pediatric MATCH Screening Trial)

Recruiting

Osteosarcoma Maintenance Therapy With OST31-164

Recruiting

Ensartinib in Treating Patients With Relapsed or Refractory Advanced Solid Tumors, Non-Hodgkin Lymphoma, or Histiocytic Disorders With ALK or ROS1 Genomic Alterations (A Pediatric MATCH Treatment Trial)

Recruiting

Chemotherapy for the Treatment of Patients With Newly Diagnosed Very Low-Risk and Low Risk Fusion Negative Rhabdomyosarcoma

Recruiting

CAMPFIRE: A Study of Ramucirumab (LY3009806) in Children and Young Adults With Desmoplastic Small Round Cell Tumor

CAMPFIRE: A Study of Ramucirumab (LY3009806) in Children and Young Adults With Synovial Sarcoma

Recruiting

A Study to Compare Early Use of Vinorelbine and Maintenance Therapy for Patients With High Risk Rhabdomyosarcoma

Recruiting

A Study of a New Drug, Nirogacestat, for Treating Desmoid Tumors That Cannot be Removed by Surgery

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CRISPR editing of the GLI1 first intron abrogates GLI1 expression and differentially alters lineage commitment.

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